HDAC Activity Is Required for Efficient Core Promoter Function at the Mouse Mammary Tumor Virus Promoter

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HDAC Activity Is Required for Efficient Core Promoter Function at the Mouse Mammary Tumor Virus Promoter

Histone deacetylases (HDACs) have been shown to be required for basal or inducible transcription at a variety of genes by poorly understood mechanisms. We demonstrated previously that HDAC inhibition rapidly repressed transcription from the mouse mammary tumor virus (MMTV) promoter by a mechanism that does not require the binding of upstream transcription factors. In the current study, we find ...

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Stably integrated mouse mammary tumor virus long terminal repeat DNA requires the octamer motifs for basal promoter activity.

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Promoter complex in the central part of the mouse mammary tumor virus long terminal repeat.

Unique among the retroviruses, mouse mammary tumor virus (MMTV) carries, in addition to the usual long terminal repeat (LTR) promoter, another promoter, P2, which is located in the central part of the proviral U3 sequence, within the LTR open reading frame (ORF). Using an in vitro reporter system based on a sensitive luciferase expression assay, we investigated the regulation of the P2 promoter...

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Functional redundancy of octamer elements in the mouse mammary tumor virus promoter.

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Evidence that nucleosomes on the mouse mammary tumor virus promoter adopt specific translational positions.

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ژورنال

عنوان ژورنال: Journal of Biomedicine and Biotechnology

سال: 2011

ISSN: 1110-7243,1110-7251

DOI: 10.1155/2011/416905